(5)Another crucial finding is that submaximal exercise resulted in a decrease in AS in each of the three groups. However there was a higher relative change in group 1( 31.58±7.
44) in comparison to group 3 ( 2.98±13.65) for submaximal.
This indicates that the higher the relative change in submaximal exercise, the lower the perceived fitness of the individual. Therefore the greater the fitness level of the individual the lower the percentage change. As a result it can be concluded regular exercise is proactive in desitizing platelet activity.
This is an adaptive measure of exercise training. Training increases prostacyclin, a powerful platelet aggregation inhibitor whilst also elevating nitric oxide which increases antiplatelet action(Mant, Kappagoda and Quinlan, 1984). A study carried out on middle aged, overweight men with mild hypertension established that low-moderate (ie submaximal) intensity was correlated with a reduction in platelet aggregation (Rauramaa et al., 1986). Eidt et al., (1989) carried out a similar study and revealed that submaximal exercise ( treadmill paced at a speed of 1mph for 30 minutes) would stimulate platelet aggregation. This was inconsistent with the findings of the study conducted. This result could have been due to the different population used in each of the studies.
This studied used males whereas Eidt et al., (1998) used dogs with coronary occlusion. As blood circulation through constricted vessels is under high stress, even the mildest form of exercise may result in extreme shear stress levels leading to aggregation. Therefore this could be a reason for the differing results in this area.Other differences in the results could be due to limitations in the study. This study was conducted in DCU and primarily the participants were recruited from the health and sports science faculty. Therefore the study only represents a limited population. A larger recruitment of very low fit individuals would have proved more significant results.
This would have advanced the finding on the correlation between platelets and exercise intensity as well as highlighting the the strong relationship with CVD. The ratio of low fit to high fit individuals wasn’t exact and therefore could have been a limiting factor. According to Faber and Fonseca (2014) sample size is a key mechanism in compromising a study.
Aerobic fitness was self assessed by the participant using a questionnaire. Due to it being self evaluation of fitness there is a level of inaccuracy to this assessment due to participants usually underestimating and overestimating self fitness or aerobic fitness levels. These levels should be measured by way of quantitative assessment.
According to Sharkey and Davis (2008) quantitative research should be used to assess differences among groups and qualitative research should be used to flesh out previous knowledge to gain a greater scope of information.